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Animal reproduction studies have shown an adverse effect on the fetus and there are no adequate and well-controlled studies in humans, but potential benefits may warrant use of the drug in pregnant women despite potential risks.
There are no controlled studies in breastfeeding women, however the risk of untoward effects to a breastfed infant is possible; or, controlled studies show only minimal non-threatening adverse effects. Drugs should be given only if the potential benefit justifies the potential risk to the infant.
Typical usage: NULL
Side Effects: Harmless alteration in the consistency, color and odor of stool may occur. A sensation of fullness, flatulence, and occasional episodes of nausea are possible during high- dosage administration. They can be avoided by administration of the enzyme preparat
Drug Interaction: is known to interact with other drugs likeAmiloride (HCl),Ascorbic Acid,Ciprofloxacin,Docetaxel,Gemifloxacin,Hydrochlorothiazide,Lomefloxacin (HCl),Spironolactone,Triamterene.
Mechanism Of Action: Rutoside is the glycoside, formed by combing a disaccharide and flavonol, it has antioxidant activity, as well as anti- inflammatory, antihistaminic and antiviral properties. Rutoside increases the strength of the walls of the blood capillaries and regulates their permeability so as to normalize pathologically increased vessel permeability.Many, if not most, of Rutoside probable actions can be accounted for, in part, by Rutoside antioxidant activity. Rutoside is a phenolic antioxidant and has been confirmed to forage superoxide radicals.. Ferrous cations are intricate in the so-called Feton reaction, which produces reactive oxygen species. Rutoside may also control the respiratory burst of neutrophils. The in vivo antioxidant action of Rutoside is most likely due to its metabolite Quercetin, to which it is converted following ingestion. Rutoside may also help sustain levels of the biological antioxidant reduced glutathione.
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Showing 5 of 18